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Attention Deficit (Hyperactivity) Disorder (ADD/ADHD)


 
Introduction Back to Top
What should I know about Attention Deficit Hyperactivity Disorder?

As any parent of a child diagnosed with attention deficit hyperactivity disorder (ADHD) can tell you, coping with the daily frustrations is a challenge at best. ADHD is one of the most frequently diagnosed childhood psychiatric condition.(1) While there are many theories regarding the cause of ADHD, we now know that many of the children diagnosed today, have a biological parent who shares the same diagnosis. Other factors that may contribute to the symptoms of ADHD are fetal alcohol syndrome, lead poisoning, meningitis, and a genetic resistance to thyroid hormone.(2, 3) Also, while not a primary cause, there seems to be a positive association that exists between negative family environment factors and ADHD.(2, 4)

We are fortunate that the diagnosis of ADHD has come as far as it has. Before 1980, a child with a short attention span who seemed impulsive and hyperactive was diagnosed with Minimal Brain Dysfunction. However, experts in the field argued that this was not an accurate diagnosis because there was no common neurologic problem that was consistent among the children diagnosed. In 1980, the American Psychiatric Association made a decision to look more at the behavioral side of the diagnosis, calling it ADD, or Attention Deficit Disorder. We now know that hyperactivity is almost always present so the name has been changed to ADHD.

What causes ADHD?

The Lancet published a study in 1985, which reported that 79 percent of hyperactive children improved when suspect foods were eliminated from their diets, only to become worse again when the foods were reintroduced. Artificial colorings and flavorings were the most serious culprits; sugar was also found to have a noticeable effect. The New York public school system initiated an experimental design in which sugar, food additives, and preservatives were gradually eliminated from the school cafeterias. During the four-year period of dietary modifications, the mean academic performance percentile rating increased from 39.2 percent to 54.9 percent.(5) Additives include artificial flavors and colors, preservatives including BHA and BHT, and sugars that can be identified in the forms of sucrose, fructose, corn syrup, mannitol, sorbitol, and other sweeteners.

Mineral status among those with ADHD has been the subject of several published clinical trials. Magnesium deficiency is the most common of the mineral deficiencies associated with ADHD. (6) In one study, magnesium deficiency was identified in 95 percent of the ADHD children examined.(7) ADHD may be influenced by dysbiosis. The presence of dysbiotic flora is encouraged by the use of antibiotics, which can destroy "friendly" or probiotic flora normally inhabiting the intestinal mucosa. The average child undergoes multiple courses of antibiotic treatment in the first five years of life, typically without replacement of probiotics. The resulting overgrowth of yeast and other pathogenic flora has been linked to alterations of immune function, food sensitivities, and ADHD. A study reported that high levels of antimetabolites, consistent with fungal or Candida related complex, were identified in the urine of children with ADHD.(8)

A deficiency in essential fatty acids (EFAs) is being singled out by some as a cause of ADHD. EFAs influence ADHD primarily in two ways: they influence gut permeability and are needed for the proper development of brain tissue.(9)

Additional Links Back to Top
  • Statistics about Attention Deficit (Hyperactivity) Disorder (ADD/ADHD)
  • Signs and Symptoms of Attention Deficit (Hyperactivity) Disorder (ADD/ADHD)
Footnotes Back to Top
1 McGough JJ, McCracken JT. Assessment of attention deficit hyperactivity disorder: a review of recent literature. Curr Opin Pediatr. Aug2000;12(4):319-24.
View Abstract
2 Cantwell CB. Attention Deficit Disorder: A review of the last 10 years. J Am Acad Child Adolesc Psychiatry. 1996;35:978-987.
View Abstract
3 Zametkin AJ. Attention Deficit Disorder: Born to be hyperactive? Grand rounds at the Clinical Center of the National Institutes of Health. JAMA. 1995;16:174-184.
4 Biederman J. Family-environmental risk factors for attention deficit hyperactivity disorder. Arch Gen Psychiatry. 1995;52:464-470.
View Abstract
5 Schoenthaler, et al. The impact of a low food additive and sucrose diet on academic performance in 803 New York City public schools. International Journal of Biosocial Research. 1986;8:185-196.
6 Starobrat-Hermelin B. The effect of deficiency of selected bioelements on hyperactivity in children with certain specified mental disorders. Ann Acad Med Stetin. 1998:297-314.
View Abstract
7 Kozielec T, Starobrat-Hermelin B. Assessment of magnesium levels in children with attention deficit hyperactivity disorder (ADHD). Magnes Res. Jun1997;10(2):143-8.
View Abstract
8 Hanna GL, Ornitz EM, Hariharan M. Urinary catecholamine excretion and behavioral differences in ADHD and normal boys. J Child Adolesc Psychopharmacol. 1996;6(1):63-73.
View Abstract
9 Burgess JR, Stevens L, Zhang W, Peck L. Long-chain polyunsaturated fatty acids in children with attention-deficit hyperactivity disorder. Am J Clin Nutr. Jan2000;71(1 Suppl):327S-30S.
View Abstract

 
About Attention Deficit (Hyperactivity) Disorder (ADD/ADHD)
Introduction
Statistics
Signs & Symptoms
Treatment Options

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